PubMed 36733839

PubMed ID: 36733839

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Mutation introduced in DDTFR10/A gene of ethylene response element-binding protein (EREBP) family through CRISPR/Cas9 genome editing confers increased Fusarium wilt tolerance in tomato.
Authors: Ijaz Siddra, Haq Imran Ul, Razzaq Hafiza Arooj
Journal: Physiology and molecular biology of plants : an international journal of functional plant biology (Physiol Mol Biol Plants), Vol.29(1), 2023‑Jan

DOI: 10.1093/emboj/16.11.3207 PMCID: PMC1169938

Abstract
We investigated the role of the DDTFR10/A gene of the ethylene response element-binding protein (EREBP) family through the CRISPR/Cas9 genome editing approach. The associated role of this gene in tomato fruit ripening was known. The involvement of ripening-regulatory proteins in plant defense has been documented; therefore, to find the involvement of the DDTFR10/A gene in host susceptibility, we introduced the mutation in DDTFR10/A gene through CRISPR/cas9 in the genome of the tomato plant. The 50% biallelic and 50% homozygous mutations were observed in the T0 generation. The CRISPR/Cas9 edited plants showed 40% reduced symptoms of Fusarium wilt compared to control plants (non-edited). The DDTFR10/A gene expression in tomato plants was evaluated against biotic (Fusarium wilt) and abiotic (salinity) stresses, and the upregulated expression of this gene was found under both challenges. However, a comparative increase in DDTFR10/A gene expression was observed in tomato plants upon inoculation with Fusarium oxysporum f. sp. lycopersici. The phenotypic assay performed on edited tomato plants demonstrated the role of the DDTFR10/A gene in contributing toward susceptibility against Fusarium wilt.

The online version contains supplementary material available at 10.1007/s12298-022-01273-6.
Publication Types
Journal Article
Keywords
CRISPR/Cas9 genome editing DDTFR10/A gene Negative regulator Salinity Tomato wilt
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